The evidence that proves that xenoestrogens are causing a dangerous imbalance on male potency and capacity to reproduce is quite abundant.
These devastating effects can begin to develop during the fetal life when the male fetus is exposed to abnormal levels of estrogens¹.
Researchers believe that the current increased incidence of testicular cancer in men is most likely due to this fetal exposure to estrogenic chemicals, which interferes with the ability of gonadal steroids to support normal tissue differentiation in utero.
Researchers in the 20th century started realizing the horrible effects of xenoestrogens.
In 1947, Charles Broley, who was an ornithologist, began to notice strange changes in the behavior of the eagles.
He realized that the number of baby eagles was decreasing sharply, and big eagles were acting weird and not showing interest in mating.
Has time passed, he was convinced without a doubt that almost 80% of Florida’s bald eagles were infertile.
Later on, researchers linked the problem to something called DDE, an estrogenic outcome of DDT (which was a commonly used pesticide for insect control in the United States until it was canceled in 1972 by the United States Environmental Protection Agency)².
In addition to this evidence, there’s a recent study that shows how DES (diethylstilbestrol ) which is one out of several estrogenic chemicals used as a pharmaceutical agent, can produce testicular cancer and malformation of the genitals.
In the 80’s the Tower Chemical Company near Orlando, Florida had a terrible accident. After it, DDE was flowing into a creek that emptied into Lake Apopka.
Later on, a scientific team was called in to investigate the decreasing amount of alligators.
One of the researchers said that they were observing sex-reversal and that at least 25% of all male alligators had a deformed penis with a reduced size².
It is quite disturbing to know that these dangerous products are so common, for example, the compound BSA that can bind to estrogen receptors in the plasma membrane (which is a biological membrane that separates the interior of all cells from the outside environment) has found to cause decreased testicular androgen production in men.
BSA is commonly used in farms to fatten the animals to increase profitability , this means that BSA is found in the meat we eat, like pork and chicken.
We also have the terrifying case of diethylstilbestrol or DES, which was the first successfully produced synthetic estrogen.
It was cheaply produced from coal tar derivatives and was also 3 times more estrogenic than natural estrogens.
According to Grey Matters³:
“In 1940, the FDA rejected the use of DES due to disturbing animal studies and an overall scientific uncertainty.
However, this did not slow DES use in humans.
Despite evidence of adverse events in animals, which included thyroid and pituitary damage, links to uterine cancer, sexual differentiation of offspring and other defects associated with sexual maturity, DES was pushed to market.”
So, even though there is enough evidence to show that xenoestrogens can bind estrogen receptors and produce rapid changes, the pharmaceutical industry still claims that xenoestrogens are generally much weaker than the naturally occurring hormone estrogen and therefore unable to compete and induce estrogenic activity.
Also, this synthetic estrogen is known to increase breast cancer risk in exposed women. It also produces different kinds of birth related adverse problems in their daughters such as spontaneous abortion, stillbirth, preterm delivery, second trimester pregnancy loss and neonatal death. Children in utero under the exposure of DES suffer from sub/infertility and cancer of reproductive tissues4.